Dr. Tar Krisztina

Telephone number (landline), extension: 
+36 52 412 345/62739
E-mail: 
Studies
1989 to 1993
Kossuth Lajos Tudományegyetem Gyakorló Gimnáziuma
1994 to 2000
Debreceni Egyetem
1995 to 2000
Biológus (Biokémia szakág)
2005
Ph. D. fokozat
Assignments / positions
2014
egyetemi adjunktus
DE ÁOK, Orvosi Vegytani Intézet
2013 to 2014
óraadó adjunktus
Long Island University, Brooklyn, NY
2010 to 2014
Research Associate and Postdoctoral Fellow
Albert Einstein College of Medicine of Yeshiva University, Bronx, NY
2007 to 2010
kutató
Biotalentum Kft, Gödöllő, Hungary
2006 to 2007
Postdoctoral Fellow
Medical University of Vienna
2005 to 2006
Postdoctoral Fellow
University of Chicago
2002 to 2004
Research Trainee
Johns Hopkins University, Baltimore, MD
2000 to 2005
Ph.D hallgató
DE ÁOK, Orvosi Vegytani Intézet
Scientific qualification / title
Ph.D.
Fields of interest
proteaszóma, PA200, fehérjelebontás, ubiquitin, mitokondrium, Huntington kór, fehérje aggregáció
Language skills
Angol
Francia
Experience abroad
2009
Franciaország
Jouy-En-Josas
INRA
2008
University of Madrid
1998
Franciaország
Rennes
Universite de Rennes I
Membership
1998
Magyar Biokémiai Társaság
Scientometric data

Publications

Scientific Publications

Number of citations: 
351
Hirsch-index: 
7
Other information

Válogatott közlemények:

Lontay B, Kiss A, Virág L, Tar K. How Do Post-Translational Modifications Influence the Pathomechanistic Landscape of Huntington's Disease? A Comprehensive Review.

Int J Mol Sci. 2020 Jun 16;21(12):4282. doi: 10.3390/ijms21124282. PMID: 32560122

 

Douida A, Batista F, Robaszkiewicz A, Boto P, Aladdin A, Szenykiv M, Czinege R, Virág L, Tar K. The proteasome activator PA200 regulates expression of genes involved in cell survival upon selective mitochondrial inhibition in neuroblastoma cells.

J Cell Mol Med. 2020 Jun;24(12):6716-6730. doi: 10.1111/jcmm.15323. Epub 2020 May 5. PMID: 32368861

 

Aladdin A, Király R, Boto P, Regdon Z, Tar K. Juvenile Huntington's Disease Skin Fibroblasts Respond with Elevated Parkin Level and Increased Proteasome Activity as a Potential Mechanism to Counterbalance the Pathological Consequences of Mutant Huntingtin Protein.

Int J Mol Sci. 2019 Oct 26;20(21):5338. doi: 10.3390/ijms20215338. PMID: 31717806

 

Alatshan A, Kovács GE, Aladdin A, Czimmerer Z, Tar K, Benkő S. All-Trans Retinoic Acid Enhances both the Signaling for Priming and the Glycolysis for Activation of NLRP3 Inflammasome in Human Macrophage.

Cells. 2020 Jul 1;9(7):1591. doi: 10.3390/cells9071591. PMID: 32630207

 

Tar K, Dange T, Yang C, Yao Y, Bulteau AL, Salcedo EF, Braigen S, Bouillaud F, Finley D, Schmidt M. Proteasomes associated with the Blm10 activator protein antagonize mitochondrial fission through degradation of the fission protein Dnm1.

J Biol Chem. 2014 Apr 25;289(17):12145-56. doi: 10.1074/jbc.M114.554105. Epub 2014 Mar 6. PMID: 24604417

 

List of publications:

 

https://scholar.google.com/citations?user=eSpA7A8AAAAJ&hl=en&oi=ao

https://tudoster.idea.unideb.hu/hu/szerzok/3296


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